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By Kenneth D Tew, Paul B Fisher

Advances in melanoma Research presents valuable info at the intriguing and fast-moving box of melanoma examine, proposing notable and unique studies on numerous topics.

  • Provides info on melanoma research
  • Outstanding and unique reviews
  • Suitable for researchers and students

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Advances in Cancer Research

Advances in melanoma examine offers important info at the intriguing and fast-moving box of melanoma study, providing remarkable and unique stories on a number of themes. offers info on melanoma researchOutstanding and unique reviewsSuitable for researchers and scholars

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Kelekar, A. (2007). Autophagy delays apoptotic death in breast cancer cells following DNA damage. Cell Death and Differentiation, 14(3), 500–510. html. Aguirre-Ghiso, J. A. (2007). Models, mechanisms and clinical evidence for cancer dormancy. Nature Reviews. Cancer, 7(11), 834–846. 1038/ nrc2256. Aita, V. , Liang, X. , Murty, V. V. V. , Pincus, D. , … Levine, B. (1999). Cloning and genomic organization of beclin 1, a candidate tumor suppressor gene on chromosome 17q21. Genomics, 59(1), 59–65.

Komatsu, M. (2011). Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells. The Journal of Cell Biology, 193(2), 275–284. 201102031. -L. (2003). TSC2 mediates cellular energy response to control cell growth and survival. Cell, 115(5), 577–590. 1016/S0092-8674 (03)00929-2. , … Yamamoto, M. (2012). Accumulation of p62/SQSTM1 is associated with poor prognosis in patients with lung adenocarcinoma. Cancer Science, 103(4), 760–766. x. , & Mizushima, N. (2008). Beclin 1 forms two distinct phosphatidylinositol 3-kinase complexes with mammalian Atg14 and UVRAG.

2015), indicating that the relationship between autophagy and the production of secreted factors may vary depending on the cellular context. , 2015). Additionally, a separate study found that in cancer stem cells, the release of exosomes, which can mediate intercellular communication, coincided with the activation of autophagy (Kumar, Gupta, Shankar, & Srivastava, 2015). , 2015). Notably, these experiments did not distinguish whether any of the differentially secreted proteins actually arose from altered autophagy-dependent secretion vs secondary changes in transcriptional or translational programs following autophagy inhibition.

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