By Mary A. Carskadon
Teenagers are one of the such a lot sleep disadvantaged populations in our society. This e-book explores the genesis and improvement of sleep styles at this part of the lifestyles span. It examines organic and cultural components that effect sleep styles, provides dangers linked to loss of sleep, and divulges the consequences of environmental components corresponding to paintings and college schedules on sleep. This research will attract psychologists and sociologists of early life who've now not but thought of the real position of sleep within the lives of our adolescence.
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Additional info for Adolescent Sleep Patterns: Biological, Social, and Psychological Influences
Here, definitive assignment of many of these changes to the pubertal sex steroid exposure (as opposed to antenatal sex steroid effects) is difficult because of reliance on animal models in which these developmental stages are not as distinct as they are in the human. Control of Pubertal Onset Identification of pubertal CNS changes that anticipate the onset of puberty requires delineation of CNS processes controlling the timing of pubertal onset. Here, several models have been proposed. Some of these and the supporting studies will be considered here by way of review.
1 1 The MSLT is a test of sleep tendency or “physiological sleepiness” developed in the 1970s by the Stanford group (Carskadon & Dement, 1977b; Richardson, Carskadon, 12 mary a. carskadon Few data exist to indicate whether homeostatic mechanisms change at the childhood-to-adolescent transition or across adolescent development. Could adolescents find it easier to stay awake later because the sleep homeostatic mechanism undergoes developmental alterations? Known developmental changes in sleep physiology that occur in adolescence are rather subtle, largely involving the infrastructure of sleep and the pattern of sleepiness-alertness.
Further, in the rat, transient stimulation with NMDA agonists is sufficient to initiate the pubertal sequence, which is then sustained even after the exogenous stimulation is removed (Ojeda, 1991). This finding is most consistent with a primary role of diminished stimulatory influence. In view of support for both factors, and the variation across animal models, Ojeda (1991) suggests that both decreased stimulation and enhanced inhibition may be working to restrain GnRH release until the onset of puberty.